Efficiency of ascorbic acid against oxidative stress-induced dimethoate toxicity in fish, Capoeta capoeta [Guldenstaedt, 1773]

dc.authoridKOC, Evren/0000-0002-0022-9433
dc.authoridERSAN, Yusuf/0000-0002-0065-3656
dc.authoridKOC, EBUBEKIR/0000-0002-9069-715X
dc.authoridTanriverdi, Erdi Anil/0000-0002-5858-1771
dc.contributor.authorYilmaz, M.
dc.contributor.authorKaya, I.
dc.contributor.authorKoc, E.
dc.contributor.authorTanriverdl, E. A.
dc.contributor.authorKaya, M. M.
dc.contributor.authorErsan, Y.
dc.date.accessioned2024-09-29T16:06:43Z
dc.date.available2024-09-29T16:06:43Z
dc.date.issued2017
dc.departmentKarabük Üniversitesien_US
dc.description.abstractAim: The main objective of the present study was to evaluate the effect of ascorbic acid against oxidative stress-induced by dimethoate in Capoeta capoeta. Methodology: The fish caught in Kars Creek were randomized in four equal groups and acclimatized in separate tanks. The fish were kept in tanks for 10 days as follows: Group I (control) was kept in normal water, Group II, Ill and IV were kept in separate tanks containing 2 mgrdimethoate and 2 mgr dimethoate plus 100 mgr' ascorbic acid and ascorbic acid. The level of total sialic acid, total antioxidant status and total oxidant status in fish serum were analyzed. Results: Total sialic acid level of Group III was found to be lower than the level of Group II (P< 0.05), there were no statistically important differences between these two groups in total antioxidant status and oxidant status. In gills, intensive cartilage degenerations and mononuclear cell infiltrations in group II and III were observed. It was found that toxic effects of dimethoate in group III decreased. Interpretation: Ascorbic acid in fish exposed to dimethoate reduced the level of total sialic acid and degenerations in lamellae of gills, it had no significant effect on the oxidative stress response. [GRAPHICS] .en_US
dc.identifier.doi10.22438/jeb/38/5/PRN-127
dc.identifier.endpage833en_US
dc.identifier.issn0254-8704
dc.identifier.issue5en_US
dc.identifier.scopus2-s2.0-85027400863en_US
dc.identifier.scopusqualityQ3en_US
dc.identifier.startpage829en_US
dc.identifier.urihttps://doi.org/10.22438/jeb/38/5/PRN-127
dc.identifier.urihttps://hdl.handle.net/20.500.14619/7010
dc.identifier.volume38en_US
dc.identifier.wosWOS:000407544700017en_US
dc.identifier.wosqualityQ4en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.language.isoenen_US
dc.publisherTriveni Enterprisesen_US
dc.relation.ispartofJournal of Environmental Biologyen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectAscorbic aciden_US
dc.subjectCapoeta capoetaen_US
dc.subjectDimethoate toxicityen_US
dc.subjectOxidative stressen_US
dc.subjectSialic aciden_US
dc.titleEfficiency of ascorbic acid against oxidative stress-induced dimethoate toxicity in fish, Capoeta capoeta [Guldenstaedt, 1773]en_US
dc.typeArticleen_US

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